In the last bulletin we discussed thedifference in molecular structure betweenCapTri and conventional fats.
Conven-tional dietary fat is very similar, chemi-cally, to body fat, so the body has a strongtendency to simply “file away” dietary fatas body fat.Conventional fats are circulatedthroughout the body, picked up by fat cellsand stored in the presence of insulin andglucose (1).
In contrast, CapTri® isnot circulated throughout the body, butinstead is transported directly from thesmall intestine to the liver. CapTri® is re-tained in the liver, where it is rapidlyburned to produce energy (2).Excess calories from any food canbe stored as body fat, but some foods aremuch easier to convert to body fat thanothers. Conventional fats are the easiestto store as body fat because they alreadyhave the same chemical structure as bodyfat. By comparison, protein and carbohy-drate have to be chemically converted intofat before they can be stored as fat.
Thisconversion process uses up some of thecalories contained in the protein or car-bohydrate food, so this reduces the ten-dency of these foods to contribute to bodyfat stores. This is why we recommend adiet high in natural complex carbohydratesand lean protein, and that consumption ofconventional fats be kept to an absoluteminimum. Just be sure to take a table-spoon of linseed oil or safflower oil ev-eryday to supply essential fatty acids.Since CapTri® is transported to theliver (instead of fat cells) and burned forenergy, it’s no surprise that it contributesless to body fat accumulation than con-ventional fats do. The amazing thing isthat CapTri® contributes less to body fatstores than carbohydrates (3, 4). Eventhough we’ve just started our discussionof how CapTri® works, we’ve already hiton the central reason why so many body-builders are using it today: CapTri® isextremely caloric dense, but due to theway it is metabolized, it has very little ten-dency to be stored as body fat.
It’s theideal supplement for bodybuilders—con-centrated source of calories that won’tmake you fat. The calories from CapTri®represent an energy source which can beused to fuel activity or support weightgain.Now, let’s examine what happenswith CapTri® inside the liver. The liver isa miraculous organ; it’s the workhorse ofyour metabolism. Your liver knows moreabout biochemistry than all of the scien-tists in the world. When you mentionbodybuilding, everybody thinks ofmuscles. But your liver is doing all thebehind-the-scenes biochemical work thatmakes it possible. If your muscles are arace car, then your liver is the pit crew.Your liver occupies a central position inmetabolism, including metabolism of fat.Once inside the cells, fat moleculesare burned in structures called mitochon-dria.
The mitochondria are little furnaceswhere the foods you eat are burned to pro-duce energy. All of the enzymes respon-sible for fat burning are located in themitochondria. Therefore, if fats are notpermitted to enter the mitochondria, theycannot be burned for energy. Conven-tional fats (LCTs) cannot simply enter themitochondria by themselves because theycan’t penetrate the mitochondrial mem-brane (1). Instead, they have to be ac-tively transported across the mitochon-drial membrane by a special transport sys-tem called the carnitine shuttle. With-out the carnitine shuttle (1), conventionaldietary fats and body fat cannot be burnedfor energy. (Given an adequate diet yourbody can make its own carnitine, but weadd some to our Lipotropic Formula justto make sure.)Carbohydrate metabolism generatesan intermediate called malonyl-CoA,which inhibits the carnitine shuttle (1).
Furthermore, carbohydrates trigger insu-lin release, and insulin stimulates fat syn-thesis and fat storage. For these reasons,fat is not burned much as an energy sourceif carbohydrate fuel is available. This iswhy we recommend that you do your aero-bics either first thing out of bed beforebreakfast, right after training or after yourlast meal because at these times you’rerelatively carb depleted and will burnmore fat for energy.In contrast to conventional fats,CapTri® can enter the mitochondria by it-self and does not require the carnitineshuttle (1, 2). Therefore CapTri® can be burned for energy even in the presence ofcarbohydrates. This is another reason whyCapTri® has very little tendency to con-tribute to body fat stores: Conventionalfats can be burned only after carbohydratefuels have been exhausted, but CapTri®can be burned at the same time as carbs(5). The calories from CapTri® are anadditional energy source which can beused at the same time as carbohydrates,and this helps the carbohydrate fuel lastlonger (6). This is known as the glycogensparing effect .
The more energy you have,the longer and harder your workouts canbe.Lets’ summarize the reasons whyCapTri® is not stored as fat:• Conventional fats have the samemolecular structure as body fat, butCapTri® has a different molecular struc-ture (1, 2).• Conventional fats are circulatedthroughout the body, and insulin stimu-lates fat cells to pick up the fat moleculesfrom the bloodstream and store them.CapTri® is not circulated throughout thebody but is processed by the liver (1, 2).• In the liver, CapTri® is rapidlyburned to produce energy. Some of thisenergy is used by the muscles and someis converted to body heat. Once theCapTri® is burned up, there’s nothing leftto be stored (2).• CapTri® does not require the car-nitine shuttle for entry into the mitochon-dria, so it can be burned for energy at thesame time as carbohydrates (2, 5).For these reasons, we recommendCapTri® as a key supplement in your nu-tritional program. Unlike dietary fats,CapTri® supplies the body with caloriesready to be burned for energy with verylittle tendency to be stored as body fat.
1. Record, Kolpeck and Rapp, Longchain versus medium chain length triglyc-erides – a review of metabolism an clinicaluse. Nutr. Clin. Prac. 1: 129-135, 1986.
2. Bach and Babayan, Mediumchain triglycerides: an update. Am. J.Clin. Nutr. 36: 950-962, (1982).
3. Lavau and Hashim. Effect ofmedium chain triglycerides on lipogenesisan body fat on the rat. J. Nutr. 108:613-620, 1978.
4. Crozier, Bois-Joyeux, Chanez,Girard and Peret, Metabolic effects in-duced by long-term feeding of mediumchain triglycerides in the rat. Metabolism36: 807-814, 1987.
5. Sucher, Medium chain triglycer-ides: a review of their internal use in clini-cal nutrition. Nutr. Clin. Prac. 44: 146-150, 1986.
6. Cotter, Taylor, Johnson andRowe, A metabolic comparison of purelong chain triglyceride lipid emulsion(LCT) an various medium chain triglyc-eride (MCT) – LCT combination emul-sions in dogs. Am. Clin. Nutr. 45: 927-939, 1987.